The Role of Fibrin in Fibromyalgia Pain
Fibromyalgia sufferers may experience pain as a result of hypercoagulation (thickened blood) that can lead to poor circulation due to an excess build-up of fibrin. Fibrin is a whitish, insoluble protein that makes up a blood clot when promoted by thrombin, a key clot promoter.
In otherwise healthy individuals, one burst of thrombin should generate enough fibrin to form an actual clot in the event of an injury. However, those with fibromyalgia and other chronic conditions may continuously generate low thrombin levels that can result in hypercoagulation and pain as circulation slows.
Reducing fibrin with the use of systemic enzyme, anti-oxidant and anti-inflammatory nutrient therapy may be an alternative to taking blood thinning and pain relieving medications for those who do suffer from fibromyalgia.
What is the role of fibrin in fibromyalgia?
Research from the late 1990s reveals that many patients with chronic disease may have an underlying coagulation defect contributing to their symptoms. When tissues don’t receive sufficient blood, the cells are starved for oxygen and nutrients. Pain can result, as in headache or fibromyalgia.
In the case of fibromyalgia, fibrin (a stiff protein) forms a meshwork, or screen that slows blood flow to the cells. This interferes with cell function and cell-to-cell communications leading to the accumulation of additional forms of protein debris.
Therefore, the symptoms of fibromyalgia can respond well to treatments that are aimed at clearing protein debris. [1]
Thank you Linda Hoggard
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